Every three seconds, someone in the world develops dementia. Alzheimer's disease is the most common form of dementia, accounting for between 60% and 70% of all cases.

Scientists have made significant progress in understanding Alzheimer's disease, but there is still no cure due to its complex causes. Two key proteins involved in the disease are amyloid-beta and tau, which form plaques and tangles in the brain, disrupting neuron communication and leading to cell death.

Recent research has discovered that a protein from Helicobacter pylori, known for causing stomach ulcers, can block the buildup of both amyloid-beta and tau. This unexpected finding may offer a new strategy in the fight against Alzheimer's.

The study found that a protein fragment from H. pylori could effectively prevent the formation of amyloid aggregates in the brain. This discovery could potentially lead to new treatments for Alzheimer's and other diseases driven by toxic protein accumulation.

While the research is still in early stages and conducted in lab settings, it opens up new possibilities for understanding the role of bacteria in diseases like Alzheimer's. It may prompt a shift towards a more precise and personalized approach to treatment in the future.